by:Aaysha Cader Whilst much has been written about COVID-19 and its inflammation-thrombosis-hypercoagulability cascade, this has to a large extent focused on venous thromboembolism (VTE). As more data emerges, it is evident that the intense inflammatory response associated with SARS-COV-2 infection may trigger thrombosis across multiple vascular beds, with large thrombus burden in ST-segment elevation myocardialContinue reading “Do COVID STEMIs have a higher thrombus burden? More questions & implications”
The Heart in the Time of COVID-19 